22 Instrumental variables
Angrist and Pischke (2008) (p.114) describe instrumental variables (IVs) as “the most powerful weapon in the arsenal” of econometric tools. Accounting researchers have long used instrumental variables in an effort to address concerns about endogeneity (Larcker and Rusticus, 2010; Lennox et al., 2012) and continue to do so.
Unfortunately, faulty reasoning about instruments is common in accounting research. Larcker and Rusticus (2010) lament that “some researchers consider the choice of instrumental variables to be a purely statistical exercise with little real economic foundation” and call for “accounting researchers … to be much more rigorous in selecting and justifying their instrumental variables.” And a review of instrumental variable applications conducted by Gow et al. (2016) suggests that accounting researchers have paid little heed to the suggestions and warnings of Larcker and Rusticus (2010), Lennox et al. (2012), and Roberts and Whited (2013).
As we shall demonstrate in this chapter, the requirements for valid instruments are demanding and the supply of plausibly valid IVs in accounting research to date is arguably zero. As such, the use of IV should be rare, and readers, editors, and reviewers should sceptically apply the requirements discussed below to any proposed IV.
The code in this chapter uses the following packages. For instructions on how to set up your computer to use the code found in this book, see Chapter 1.2.1.
library(dplyr, warn.conflicts = FALSE)
library(MASS) # For mvrnorm()
library(AER) # For ivreg()
library(stargazer)
We use the stargazer
package to produce neat output from regressions.
For the HTML version of this book, we set sg_format
to "html"
, but "text"
would be a better option if looking at the results interactively.
sg_format < "html"
22.1 The canonical causal diagram
Gow et al. (2016) argue that evaluating instrumental variables requires careful theoretical causal, rather than statistical, reasoning. Statistical reasoning is what you will find in most econometrics textbooks, which discuss requirements regarding probability limits (e.g., \(\mathrm{plim}\;Z^{\mathsf{T}}X \neq 0\) and \(\mathrm{plim} \;Z^{\mathsf{T}}\epsilon = 0\)). In practice, you are more likely to see researchers invoking ideas such as the “only through” criterion, which are better understood as causal ideas.
Causal diagrams provide a useful framework for highlighting the requirements for a valid instrument and thus (hopefully) avoiding incorrect reasoning. The causal diagram below can be viewed as the “canonical” causal diagram for instrumental variables.
This causal diagram is similar to the “\(Z\) is a confounder” figure we saw in Chapter 5, but for two differences. First, the confounding variable \(U\) is not observed. (What was labelled \(Z\) in before is now labelled \(U\) because of the convention of using \(Z\) for instrumental variables, which we follow here.) Our reasoning in Chapter 5 suggested that if we don’t condition on \(U\), the coefficients obtained from a regression of \(Y\) on \(X\) will be biased estimates of the causal effect of \(Y\) on \(X\). But with \(U\) unobserved, we simply cannot use a strategy of conditioning on \(U\) to produce valid causal inferences.
The second difference is that we have a new variable \(Z\), which is our instrumental variable. It turns out that a variable with the attributes that \(Z\) has in the causal diagram will (subject to certain additional technical requirements) allow us to get consistent estimates of the causal effect of \(X\) on \(Y\).
This causal diagram implies three critical features of \(Z\). First, \(Z\) is random. This is implied by the absence of any arrows into \(Z\). Second, \(Z\) has a causal effect on \(X\), which is indicated by the arrow from \(Z\) to \(X\). (Later we will see that the strength of the causal relation between \(Z\) and \(X\) is important for reliable inferences.) Third, there is no direct causal effect of \(Z\) on any variable other than \(X\). This is the “only through” criterion we mentioned earlier: the effect (if any) of \(Z\) on \(Y\) goes “only through” \(X\).
Consistent with the causal perspective we take here, Angrist and Pischke (2008, p. 117) argue that “good instruments come from a combination of institutional knowledge and ideas about the process determining the variable of interest.” One study that illustrates this is Angrist (1990). In that setting, the draft lottery is well understood as random and the process of mapping from the lottery to draft eligibility is well understood. Furthermore, there are good reasons to believe that the draft lottery does not affect anything else directly except for draft eligibility.^{159}
It is perhaps worth reiterating that arguing that the only effect of an instrument on the outcome variable of interest is via the treatment of interest does not suffice to establish a valid instrument. Even if the claim that \(Z\) only affects \(Y\) via its effect on \(X\) is true, the researcher also needs to argue that variation in the instrument (\(Z\)) is asif random. For example, if \(Z\) is a function of a variable \(W\) that is also associated with \(Y\) then even if the “only through” criterion is satisfied (i.e., the only effect of \(Z\) on \(Y\) occurs via \(X\)), IV estimates of the effect of \(X\) on \(Y\) will in general be biased.^{160}
Unfortunately, there are few (if any) accounting variables that meet the requirement that they randomly assign observations to treatments and do not affect the outcome of interest outside of effects on the treatment variable. Sometimes researchers turn to lagged values of endogenous variables or industry averages as instruments, but these too are problematic.^{161}
22.2 Estimation
We now examine the estimation of IV in detail. Suppose that the outcome variable is generated by the following equation
\[y = X \beta + \epsilon\]
where \(X\) is an \(n \times k\) matrix and \(\beta\) is a \(k\)element vector of coefficients.
The goal of a researcher is to estimate \(\beta\), which provides the causal effect of \(X\) on \(y\). The OLS estimator is
\[ \widehat{\beta}_\mathrm{OLS} = (X^{\mathsf{T}} X)^{1} X^{\mathsf{T}} y = (X^{\mathsf{T}} X)^{1} X^{\mathsf{T}} (X \beta + \epsilon) = \beta + (X^{\mathsf{T}} X)^{1} X^{\mathsf{T}} \epsilon\]
When \(X\) and the other unmeasured, causal variables collapsed into the \(\epsilon\) term are correlated, the OLS estimator is generally biased and inconsistent for \(\beta\).
Note that the predicted values from an OLS regression are calculated as
\[ \widehat{y} =X \widehat{\beta}_\mathrm{OLS} = X (X^{\mathsf{T}} X)^{1} X^{\mathsf{T}} y = P_X y\] Where \(P_X\) is defined as
\[ P_X =X(X^{\mathsf{T}} X)^{1}X^{\mathsf{T}}\] we can express the predicted values as
\[ \widehat{y} = X \widehat{\beta} = P_Z y\] \(P_X\) is an example of a projection matrix. For a matrix \(W\) and a vector \(v\), the multiple of the projection matrix \(P_W\) and \(v\) projects \(v\) into the linear subspace defined by \(W\). If \(W\) is an \(n \times k\) matrix, then \(P_W\) will be an \(n \times n\) matrix that is symmetric (meaning that \(P_X^{\mathsf{T}} = P_X\)) and idempotent (meaning that \(P_W \times P_W = P_W\)).
Suppose we had a set of \(r\) variables \(Z\) comprising both exogenous elements of \(X\) and instrumental variables where \(r \geq k\).
We can use \(Z\) to calculate the twostage least squares (2SLS) estimator as
\[ \widehat{\beta}_\mathrm{2SLS} = (X^{\mathsf{T}} P_Z X)^{1}X^{\mathsf{T}} P_Z y,\] Denoting \(\hat{X}\) as \(P_Z X\), we could regress \(y\) on \(\widehat{X}\) using OLS to get the 2SLS estimator.
\[ \begin{aligned} \hat{\beta} &= (\hat{X}^{\mathsf{T}} \hat{X})^{1} \hat{X}^{\mathsf{T}} y \\ &= \left((P_Z X)^{\mathsf{T}}P_Z X \right)^{1} (P_Z X)^{\mathsf{T}} y \\ &= \left(X^{\mathsf{T}} P_Z^{\mathsf{T}} P_Z X \right)^{1} (P_Z X)^{\mathsf{T}} y \\ &= \left(X^{\mathsf{T}} P_Z X \right)^{1} X^{\mathsf{T}} P_Z y \\ \end{aligned} \] (Note that we used the idempotency and symmetry of \(P_Z\) in this analysis.)
We can rewrite the underlying model as
\[ y = \hat{X} \beta + (X  \hat{X}) \beta + \epsilon \] We can think of \(\widehat{\beta}_\mathrm{2SLS}\) as the estimator from this model (i.e., from regressing \(y\) on \(\widehat{X}\)) with an error term equal to \((X  \hat{X}) \beta + \epsilon\).
Here \(\hat{X}\) and \((X  \hat{X})\) are respectively the fitted value and residual from an OLS regression of \(X\) on \(Z\), so \(\hat{X}^{\mathsf{T}} (X  \hat{X}) = 0\) and \(\operatorname{plim} N^{1} \hat{X}^{\mathsf{T}} (X  \hat{X}) \beta = 0\).
Given that \[ N^{1} \hat{X}^{\mathsf{T}} \epsilon = N^{1} X^{\mathsf{T}} P_Z \epsilon = N^{1} X^{\mathsf{T}} Z (N^{1} Z^{\mathsf{T}} Z)^{1} N^{1} Z^{\mathsf{T}} \epsilon \] we have \(\operatorname{plim} N^{1} \hat{X}^{\mathsf{T}} \epsilon = 0\) so long as \(\operatorname{plim} N^{1} Z^{\mathsf{T}} \epsilon = 0\), which is implied by our causal diagram above. This shows that \(\widehat{\beta}_\mathrm{2SLS}\) is a consistent estimator for \(\beta\). This result depends heavily on the linearity of the model and does not generalize to nonlinear models.^{162}
A careful reader might note that we have not used the causal diagram from the previous subsection in this analysis. A heuristic analysis of the 2SLS estimator given the canonical causal diagram can be found in Cunningham (2021, pp. 323–329).
22.3 Reasoning about instruments
Larcker and Rusticus (2010, p. 189) find that in a typical accounting research paper, “there is almost no discussion regarding the choice of speciﬁc variables for instruments (only slightly more than half of the papers discuss the instruments at all). Researchers do not rigorously discuss why the variables selected as instruments are assumed to be exogenous … nearly 80% of the papers provide no justiﬁcation for the choice of instrument whatsoever.”
But, even when researchers do attempt to evaluate instruments, confusion and loose reasoning seems common. For example, in the rare cases where instruments are justified, many appear to focus on what Roberts and Whited (2013, p. 514) describe as “the question one should always ask of a potential instrument [namely] does the instrument affect the outcome only via its effect on the endogenous regressor?” But a focus on this question cannot come at the expense of discussion of the requirement that the instrument be (asif) random.
The discussion of instrument validity in Roberts and Whited (2013, p. 514) actually provides a good case study for accounting researchers, as it is a paper written to enhance reasoning about endogeneity in finance, a field closely related to accounting research.
The setting explored by Roberts and Whited (2013) is that of Bennedsen et al. (2007), who “find that family successions have a large negative causal impact on firm performance” [p. 647]. While Roberts and Whited (2013) proceed with fairly loose verbal reasoning, we think it is helpful to recapitulate their analysis using a series of questions implied by the requirements for a valid instrument.
One baseline requirement for this analysis is that we need to have a clear idea of what the instrument is, what the treatment variable is, and what the outcome is.
The quoted sentence from Bennedsen et al. (2007) identifies the treatment (family succession) and the outcome of interest (firm performance). The instrument used by Bennedsen et al. (2007) is the gender of the firstborn child of a departing CEO.
A second critical element of this analysis is that we use causal reasoning rather than statistical reasoning. While an underlying causal model will imply statistical relations, it is problematic to substitute statistical analysis of observed data for such causal reasoning.
Here are the questions:
Is the instrument (asif) random? The question is addressed quite directly by Bennedsen et al. (2007). Our knowledge of human biology tells us that the sex of a child is essentially random at time of conception. And, as “over 80 percent of firstchild births [in the sample] occurred prior to 1980, before current techniques to identify the gender of children were widespread”, there is little reason to doubt that the sex of live births isn’t similarly random.

Does the instrument have a direct causal effect on the treatment variable? Here Bennedsen et al. (2007) rely on statistical evidence: “We show that the gender of the firstborn child of a departing CEO is strongly correlated with the decision to appoint a family CEO: The frequency of family transitions is 29.4 percent when the firstborn child is female and increases to 39 percent (a 32.7 percent increase) when the firstborn is male.” But it is important that the link is causal. A merely statistical association between \(Z\) and \(X\) is problematic if we do not have a clear causal explanation. If \(X\) and \(Z\) are associated for unexplained reasons, it is difficult to rule out the possibility that those unexplained reasons include variables that drive both \(X\) and \(Z\) or effects of \(Z\) on variables other than \(X\) that might be associated with \(Y\). Either of these alternatives would undermine the validity of \(Z\) as an instrument and thus need to be ruled out.
What Bennedsen et al. (2007) appear to need here is that, for a proportion of the firms in the sample, family succession is caused by the firstborn child being a son rather than a daughter. The paper mentions “primogeniture” a couple of times and perhaps we can assume that this mechanism exists based on general knowledge of Danish customs, but more discussion beyond the statistics might have been helpful for readers with little knowledge of Danish family dynamics beyond what is depicted in Hamlet.

Does the instrument affect the outcome only through its effect on the treatment variable? Again Bennedsen et al. (2007) rely on statistical evidence: “We find that firms’ profitability, age, and size do not differ statistically as a function of the gender of the first child. Moreover, the family characteristics of departing CEOs are comparable.” But it is not possible to test the “only through” assumption statistically, instead we need “compelling arguments relying on economic theory and a deep understanding of the relevant institutional details” (Roberts and Whited, 2013, p. 515).
In this regard, the requirements are steep and Bennedsen et al. (2007) arguably fall short. One reason to doubt the “only through” requirement is the sheer amount of time between assignment of the instrument and the assignment of treatment (family succession) and the presumed preference for firstborn sons to take over. There might be 40 or 50 or more years between the birth of the first child and CEO succession. The “only through” requirements means that the sex of the first child does not affect anything about the firm other than family succession. This means that nothing about the management of the firm prior to CEO succession is affected by the increased likelihood that the firm will continue to be managed by the family after the CEO leaves. This rules out management assignment decisions that are intended to groom a family member for a CEO role, as these could affect performance through channels other than the mere fact of family succession. It also rules out decisions about the structure or performance of the firm (e.g., which portions of the firm are retained and how much effort is exerted in running it) prior to succession.
In summary, Roberts and Whited (2013) explicitly identify only Question 3 as the critical one, but in a setting where it is Questions 1 and 2 that appear to have the correct answer. But focusing on Question 3 and ignoring Question 1 can be problematic in practice, as we shall see in the next subsection.
An important caveat to the somewhat negative evaluation of the reasoning in Bennedsen et al. (2007) is that just because there are holes in the arguments for an instrument’s validity does not mean that one cannot make causal inferences. It just means that the causal reasoning has to be more circumspect and rule out alternative causal pathways from the (plausibly random) instrument to the outcome.
22.4 “Bulletproof” instruments
While some accounting researchers appear to believe that statistical tests can be used to evaluate whether an instrument is “valid” (see Larcker and Rusticus, 2010), there are no simple statistical tests for the validity of instruments. Indeed, many studies choose to test the validity of their instrumental variables using statistical tests, but such tests of instruments are of little value in practice.
A sceptical reviewer might ask us to justify our generated instruments beyond the “only through” argument we made above. For example, it is common to ask whether our instruments pass a test of overidentifying restrictions. Such tests apply when we have more instruments than endogenous variables for which we need instruments and typically posit that at least one instrument is valid and ask whether we can reject the null hypothesis that the other instruments are valid.
The sceptical reviewer might also ask us to provide evidence that we do not have weak instruments.^{163} Weak instruments lead to bias in small samples (Stock et al., 2002).
In this section, we examine what we might “achieve” if we relax the requirement for an instrument to be random (i.e., we skip Question 1 above) and rely on statistical tests to address the concerns raised by this hypothetical reviewer.
Suppose that we have \(y = X \beta + \epsilon\), with \(\rho(X, \epsilon) = 0.2\) and \(\beta = 0\). This means that there is in fact no causal relation between \(X\) and \(y\), but we have an endogeneity issue and thus might despair our inability to get good inferences about the causal relation between \(X\) and \(y\).
But, wait. We have a “solution”! Simply create three instruments: \(z_1 = x + \eta_1\), \(z_2 = \eta_2\), and \(z_3 = \eta_3\), where \(\sigma_{\eta_1} = \sigma_{\eta_2} = \sigma_{\eta_3} \sim N(0, 0.09)\) and independent. We can even make an R function to do this:
generate_ivs < function(df, sd_eta = 0.3) {
df %>%
mutate(z_1 = X + rnorm(n, sd= sd_eta),
z_2 = rnorm(n, sd = sd_eta),
z_3 = rnorm(n, sd = sd_eta))
}
Now, do these instruments pass the “only through” requirement? Clearly there is no relation between any of the instruments and any other variable except for \(z_1\), which is only associated with other variables through its relation with \(X\). So, “yes”!
What happens when we estimate IV using these instruments? We first set the parameters at the values discussed above then generate some data using the model outlined above. Here we have \(X\) and \(\epsilon\) that are bivariatenormally distributed with mean \(0\) and variance \(1\).
# Parameters for generating simulated data
n < 1000
beta < 0
corr < 0.2
Sigma < matrix(c(1, corr, corr, 1), nrow = 2)
# Used to generate instruments
sd_eta < 0.3
# Used for evaluation of IV
p.cutoff < 0.05
F.threshold < 30
# Number of simulations (used later)
k < 1000
generate_data < function() {
as_tibble(mvrnorm(n, mu = c(0, 0), Sigma = Sigma),
.name_repair = ~ c("X", "e")) %>%
mutate(y = X * beta + e)
}
set.seed(2019)
df < generate_data()
Now we run some regressions.
We first run an OLS regression.
We then generate some instruments (the generate_ivs
function could be used with any data set having an X
that we’d like to instrument for) and then estimate an IV regression.^{164}
ols < lm(y ~ X, data = df)
df_ivs < generate_ivs(df)
iv < ivreg(y ~ X  z_1 + z_2 + z_3, data = df_ivs)
Dependent variable:  
y  
OLS  instrumental  
variable  
(1)  (2)  
X  0.184^{***}  0.190^{***} 
(0.031)  (0.032)  
Constant  0.054^{*}  0.054^{*} 
(0.031)  (0.031)  
Observations  1,000  1,000 
R^{2}  0.035  0.035 
Adjusted R^{2}  0.034  0.034 
Residual Std. Error (df = 998)  0.983  0.983 
F Statistic  36.258^{***} (df = 1; 998)  
Note:  ^{}p<0.1; ^{}p<0.05; ^{}p<0.01 
In this scenario, we know that the true \(\beta\) is zero; in practice, we would not know this.
We can extract statistics related to standard tests using the summary
function.
iv.sum < summary(iv, diagnostics=TRUE)
Sargan.stat < iv.sum$diagnostics["Sargan", "statistic"]
Sargan.p < iv.sum$diagnostics["Sargan", "pvalue"]
F.stat < iv.sum$diagnostics["Weak instruments", "statistic"]
F.p < iv.sum$diagnostics["Weak instruments", "pvalue"]
We have a Sargan test statistic of 1.8 (\(p\)value of 0.41). So we “pass” the test of overidentifying restrictions. Based on a test statistic of 30, which easily exceeds the thresholds suggested by Stock et al. (2002), the null hypothesis of weak instruments is rejected as we have an \(F\)stat of 3884. So we can report to our skeptical reviewer that our instruments are “good”. And we have “results”. Yay!
But maybe we got lucky. To test this, we use the following function to see if this pattern holds across repeated runs.
run_simulation < function(run) {
df <
generate_data() %>%
generate_ivs()
iv < ivreg(y ~ X  z_1 + z_2 + z_3, data = df)
iv.sum < summary(iv, diagnostics=TRUE)
return(
tibble(
run = run,
coeff = iv.sum$coefficients[2, 1],
p.value = iv.sum$coefficients[2, 4],
Sargan.stat = iv.sum$diagnostics["Sargan", "statistic"],
Sargan.p = iv.sum$diagnostics["Sargan", "pvalue"],
F.stat = iv.sum$diagnostics["Weak instruments", "statistic"],
F.p = iv.sum$diagnostics["Weak instruments", "pvalue"]))
}
The following code runs 1000 simulations and extracts some test statistics.
sim_results < bind_rows(lapply(1:k, run_simulation))
coeff < mean(sim_results$coeff)
sig.percent < mean(sim_results$p.value < p.cutoff) * 100
F.stat < mean(sim_results$F.stat > F.threshold) * 100
reject.endogeneity < mean(sim_results$Sargan.p >= p.cutoff) * 100
The results of our simulation analysis are as follows:
 The mean estimated coefficient on \(X\) is 0.2, which is statistically significant at the 5% level 100% of the time. Note that this coefficient is close to \(\rho(X, \epsilon) = 0.2\) , which is to be expected given how our data were generated.
 Based on a test statistic of 30, which easily exceeds the thresholds suggested by Stock et al. (2002), the null hypothesis of weak instruments is rejected 100% of the time.
 The test of overidentifying restrictions fails to reject a null hypothesis of valid instruments (at the 5% level) 92.9% of the time.
Again, hooray! Our hypothetical referee should be satisfied in almost every run of the simulation. But we know that these results are bogus. The problem is that the instrument \(Z\) is not random; it’s a function of \(X\) and essentially just as endogenous as \(X\) is.
While the generated instruments we used above might seem a bit fanciful, their properties are probably not very different from such instruments as lagged values or industry averages (\(z_1\)) or random items plucked from Compustat (more like \(z_2\) or \(z_3\)), which are not unheard of in actual research.
This example shows that it is quite possible for completely spurious instruments to deliver bad inferences, yet easily pass tests for weak instruments and tests of overidentifying restrictions.
22.5 Causal diagrams: An application
To illustrate the application of causal diagrams to the evaluation of instrumental variables, we consider Armstrong et al. (2013). Armstrong et al. (2013) study the effect of shareholder voting (\(\textit{Shareholder support}_{t}\)) on future executive compensation (\(\textit{Comp}_{t+1}\)) . Because of the plausible existence of unobserved confounding variables that affect both future compensation and shareholder support, a simple regression of \(\textit{Comp}_{t+1}\) on \(\textit{Shareholder support}_{t}\) and controls would not allow Armstrong et al. (2013) to obtain an unbiased or consistent estimate of the causal relation.
Among other analyses, Armstrong et al. (2013) use an instrumental variable to estimate the causal relation of interest. Armstrong et al. (2013) claim that their instrument is valid. Their reasoning is represented graphically in Figure 22.1. By conditioning on \(\textit{Comp}_{t1}\) and using Institutional Shareholder Services (ISS) recommendations as an instrument, Armstrong et al. (2013) argue that they can identify a consistent estimate of the causal effect of shareholder voting on \(\textit{Comp}_{t+1}\), even though there is an unobserved confounder, namely determinants of future compensation observed by shareholders, but not the researcher.^{165}
While the authors note this possibility: “validity of this instrument depends on ISS recommendations not having an influence on future compensation decisions conditional on shareholder support (i.e., firms listen to their shareholders, with ISS having only an indirect impact on corporate policies through its influence on shareholders’ voting decisions)”, they are unable to test the assumption (Armstrong et al. (2013), p. 912). Unfortunately, this assumption seems inconsistent with the findings of Gow et al. (2013), who provide evidence that firms calibrate compensation plans (i.e., factors that directly affect \(\textit{Comp}_{t+1}\)) to comply with ISS’s policies so as to get a favourable recommendation from ISS. As depicted in Figure 22.2, this implies a backdoor path from \(\textit{Comp}_{t+1}\) into \(\textit{ISS recommendation}_t\), suggesting that the instrument of Armstrong et al. (2013) is not valid.^{166}
The careful reader will not that we did not need to address the randomness of the instrument (i.e., “Question 1” from above) to conclude that the instrument is problematic. It is implicit in the first causal diagram that \(\textit{ISS recommendation}_t\) is random conditional on \(\textit{Comp}_{t1}\), which would need to be included in the regression even based on the first causal diagram. A fuller analysis of the first causal diagram is beyond the scope of the current version of this course, as it would require results not covered herein. More on instrumental variables and causal diagrams can be found in Chapter 7 of Pearl (2009b).
22.6 Further reading
Given the difficulty of identifying credible instruments in accounting research, we have eschewed the replication of actual papers in this chapter. The challenges with IVs seem more apparent with idealized settings provided by simulation analysis. We direct readers interested in textbook treatments of IVs at a similar or slightly more advanced level to Cunningham (2021) and Adams (2020), each of which provides an uptodate treatment of issues related to IV estimation.
22.7 Discussion questions
You might find this template helpful for answering the following questions.
 In the 1960s and early 1970s, American men were at risk of being drafted for military service. Each year from 1970 to 1972, draft eligibility was prioritized based on the results of a lottery over birthdays. A random number was assigned to the birthdays of 19yearolds. Those whose birthday was associated with a random number below the cutoff were drafteligible; the other 19yearolds were not drafteligible. Angrist and Pischke (2008) point out that, “in practice, many drafteligible men were still exempted from service for health or other reasons, while many men who were draftexempt nevertheless volunteered for service.” Using an indicator for draft lottery number being below the cutoff as an instrumental variable, Angrist (1990) finds that those who served in Vietnam earned significantly less than their peers even in 1981. Try to draw the causal diagram for this analysis and apply the three questions outlined above to this setting. What assumptions are needed for the answer to each question to be “yes”? How might these assumptions be violated?
 Ahern and Dittmar (2012) study a rule requiring firms in Norway to appoint female directors “use the prequota crosssectional variation in female board representation to instrument for exogenous changes to corporate boards following the quota.” What is the role of the discussion on p.145 suggesting that “the quota was implemented without the consent of business leaders”? What about the discussion on p.155 suggesting that the rule was not anticipated?
 You and a coauthor are interested in studying the effect of independent directors on financial reporting quality. You conjecture that independent directors demand better financial reporting, as they are less likely to benefit from obfuscation of performance than nonindependent directors, who are often employees of the firm. However, you are concerned that causation may run in the opposite direction; firms with better financial reporting quality may have more independent directors on their boards. Provide some arguments for the existence of reverse causation. How persuasive do you find these arguments?
 In response to a request by the SEC in February 2002, the major US exchanges (NYSE and Nasdaq) proposed changes to listing standards that would require firms to have a majority of independent directors. In 2003, the SEC approved these changes and required that firms comply by the earlier of (i) the first annual shareholder meeting after January 15, 2004 and (ii) October 31, 2004. Your coauthor argues that this change was “exogenous”, as it was not driven by the decisions of individual firms, and this fact allows you to estimate a causal effect.
 Does “exogeneity” as used by your coauthor mean the same thing as exogeneity for the purposes of econometric analysis? Does the former notion imply the latter? Think of “examples” to support your arguments (examples could include a simple model, a numerical argument, or purely verbal descriptions of real or imagined scenarios).
 More specifically, your coauthor argues that the change in the number of independent directors imposed on the firm by the new rules is exogenous and thus could be used as an instrument for your study.
Armstrong et al. (2014) examine the changes in listing standards above and use “the minimum required percentage change in independent directors,
Min % change ID
, as an instrument” in studying the effect of independent directors on firm transparency. What are the parallels between the setting of Armstrong et al. (2014) and that of Ahern and Dittmar (2016)? Do the two papers use the same basic approach, or are there important differences between their approaches?
 Suppose the existence of a world of Sneetches.^{167}
There are two kinds of Sneetches:
Now, the StarBelly Sneetches
Had bellies with stars.
The PlainBelly Sneetches
Had none upon thars.
In the Sneetch world, there are 10000 (n
) firms all boards of directors have 10 (max_d
) directors.
The number of PlainBelly directors is determined by firms to maximize the function
\[v(x) = a x^2 + b x \]
where \(a = 1\) and \(b = \gamma z\), \(\gamma = 3\) and \(z\) is lognormally distributed with default parameters of \(\mu=0\) and \(\sigma=1\).
Here is the code for these parameters:
 Expressed in terms of \(a\) and \(b\), what value of \(x\) maximizes \(v(x)\)? What issue do we have in using this maximum to selected the number of PlainBelly directors on a board? What does the function below do? Does it address the issue associated with simply maximizing \(v(x)\)
Suppose that the value of firm \(i\) at time \(t=0\) is given by the equation:
\[ V_{i0} = \alpha + \beta \times b_i + \epsilon_0 \]
where \(\alpha = 100\), \(\beta = 10\) and \(\epsilon \sim N(0, \sigma_{\epsilon})\), where \(\sigma_{\epsilon} = 5\).
The following code implements this value function and calculates the value of firms at time \(t=0\)
The following code generates the value (V_0
) and the number of PlainBelly directors (d_0
) for each of the 10,000 firms at time \(t=0\) assuming that each firm chooses the number of PlainBelly directors to maximize \(v\).

Now, suppose that the Sneetches pass legislation requiring every board to have at least three PlainBelly directors at time \(t=1\).
The value of the firm \(i\) at time \(t=1\) is given by the equation:
\[ V_{i1} = \alpha + \beta \times b_i + \epsilon_{i1} \]
The following code implements this.
Note that the code adds data for \(t=1\) to the \(t=0\) values from above and that the maximization of \(v\) is subject to the constraint that \(x \geq 3\).
 Using OLS, estimate the relationship between firm value at time \(t=1\) (\(V_1\)) and the number of PlainBelly directors on the board (\(d_1\)).
 Estimate an IV regression using the strategy of Ahern and Dittmar (2012).
 Estimate an IV regression using the strategy of Armstrong et al. (2014) (note that rather than
Min % change X
you might use something likeMin change X
, as the denominator is 10 for all observations). Hint: Thepmin
function in R gives the pointwise minimum of two vectors of values (e.g.,pmin(x, y)
will return the lower of \(x\) and \(y\) for each pair.)  What is the true causal effect of PlainBelly directors on firm value? Do the strategies of either Ahern and Dittmar (2012) or Armstrong et al. (2014) correctly estimate this effect?
 The objective of Cohen et al. (2013) “is to investigate how governance regulations in SOX and the exchanges are associated with chief executive officers’ incentives and risktaking behavior.” What is the treatment of interest in Cohen et al. (2013)? What are the outcomes of interest? Cohen et al. (2013) state that “our dependent variables, namely, investment and executive incentive compensation, are likely to be determined jointly. As such, the parameter estimates from ordinary least squares (OLS) are likely to be biased. Our empirical analyses address the issue by using simultaneous equations models.” Looking at Panel A of Table 4, what “exclusion restrictions” are assumed? What does the implied causal diagram look like? How persuasive do you find the empirical approach of Cohen et al. (2013) to be?